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Alopecia areata is noncontagious or otherwise not contagious. It occurs more frequently in those who have affected members of the family suggesting that heredity is often a cause. Strong evidence that genes may increase peril for alopecia areata is discovered by studying families with 2 or more affected members. This study identified no less than 4 regions in the genome which can be likely to contain alopecia areata genes. In addition it is somewhat more planning to occur in those who have relatives with auto immune diseases.

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The condition is assumed to be an autoimmune disorder the location where the natural object attacks its hair follicles and suppresses or stops growth of hair. There is evidence that T cell lymphocytes cluster around these follicles causing excitement and subsequent hair thinning. An unknown environmental trigger including emotional stress or possibly a pathogen is thought to combine with hereditary factors to cause the condition. At that place there are some recorded cases of babies being born with congenital alopecia areata, but these are not cases of autoimmune disease because a child is born without having a fully developed immune system.
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